NAEH is a frequent clinical picture that is generally diagnosed -in a casual form- during the ecosonographic examination of the abdomen ( hiperecogenic liver) for the evaluation of different pathologies or in routine.

The pathogenic mechanism by which NAEH may develop, has not been completely explained, nevertheless, it is suspected that it can intrinsically be caused by originated metabolic alterations within the individual's own hepatic cell, or as a result of an excessive contribution of fat and/or carbohydrates to the liver, which surpass their excretory capacity.

We do not have to correlate solely to alcohol consumption the cause of this disease, we must also consider metabolic diseases, such as: obesity, alteration in lipids' metabolism (cholesterol and triacilglicerides ), diabetes mellitus types I and II, and to those patients who suffer insulin resistance.

There is strong evidence which ties NAEH to the well-known 'metabolic syndrome' also called ' syndrome X' to the point were NAEH is accepted as an additional element, associated with diabetes, hypertension, hipertrigliceridemia and obesity. Apparently, insulin resistance is the common denominator in these diseases.

The clinical examination of patients, who present/display abnormal transaminase levels, would have to take in account NAEH, especially if the person is obese or diabetic. Even though there is an important evolution in this field, the only actual form of differentiating NAEH of other liver diseases, is to carry out a hepatic biopsy.

The evolution of the EHNA is generally benign, although in some cases there is the risk that it might develop into cirrhosis. Its clinical course is long and slow and an important number of patients undergo complications, like hepatocelular carcinoma; and many of them will require liver transplant.

There is no specific synthetic treatment for this disease. Each therapeutic regime would have to include a gradual and supervised body weight reduction, a balanced diet, exercise, suspension of alcohol consumption, and the treatment of the precipitant and concomitant factors. It will have to, in a fundamental way, correct the preexisting metabolic disorder that precipitated the disease, and the hepatic alteration, both function (normalization of hepatic tests), and anatomical structure.

The treatment with Adaptógenos, formulated under the optics of Systemic Medicine has obtained the recovery of our patients with NAEH, as demonstrated by the improvement of the hepatic functionalism tests and fat ecography follow-up.

Scientific literature covers extensively the demonstrated benefits of Silybum marianum's (milk thistle) active principles and Schizandra chinensis , which have capacity to avoid and revert damage to the hepatic structure, suggesting its preventive and curative use. They are powerful hepatoprotectors, regenerators of the hepatic tissue; diminish the oxidative damage caused by free radicals, inhibiting lipidic peroxidation and damage to the hepatocyte's cellular membrane, improving metabolism; as well as the transportation and storage of fats at the hepatic level.  In addition, they prevent toxic substances from injuring liver cells. It is of great importance to remember that diseases have their causes and consequences, the indicated treatment must attack both factors, without generating new consequences, uniting all the available medical knowledge in the use of therapeutic superior plants tells (adaptogens), will help us mainly in this pathology, to restore the individual's health.